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Tuesday, September 26, 2017 2:46 AM | Lori Batchelor Volg link
At the CNHS conference, Dr. Sclafani discussed his feelings about the incomplete UBC trial on CCSVI that prematurely released negative results in March, 2017.
Lori Batchelor
Monday, September 25, 2017 7:35 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
THE OXIDASE ACTIVITY OF SERUM CERULOPLASMIN IN MULTIPLE SCLEROSIS, The Psychoneurological Institute, Pruszkow, Poland. 1966
http://onlinelibrary.wiley.com/doi/10.1111/j.1600-0404.1966.tb01190.x/full#references

Transcript: Acta Neurol. Scandinav. 42, 379-384, 1966. From the Department of Neurology and Biochemical Laboratory of the Psychoneurological Insti tute in Pruszkow, Poland. THE OXIDASE ACTIVlTY OF SERUM CERULOPLASMIN IN MULTIPLE SCLEROSIS WOJCIECH CENDROWSKI and WACLAWA SZAJBEL INTRODUCTION Enzymatic activity of a number of oxidases in white matter, cerc- brospinal fluid and serum of multiple sclerotics is still a subject of research, because of its eventually significant role in the pathogenesis of the disease. Among these enzymes appears ceruloplasmin, a metalo- proteid with molecular weight 151.000, which occurs in serum alfaz- globulins and contains 0,34 per cent of copper, Abood et a f . (19571, Deufsch (1964), Houchin (1958), Markowifz e f a[ . (1955), Morel1 & Scheinberg (1960), Osaki e f al. (1964) and others. According to Jamie- son ( 1965), structural features of human ceruloplasmin adhere more closely to the pattern found in the majority of glycoproteins. This en- zyme catalyses the oxidation of poly-phenols, diamines and ascorbic acid. Some compounds as CN-, N3- and OCN- have inhibitory effects on the activity of ceruloplasmin. The biological function of ceruloplasmin is not completely understood although possibly it takes part in the ex- change of copper between the blood stream and different tissues. Since previous publications on the level of serum ceruloplasmin in multiple sclerosis are contradictory, dysproteinemia is a frequent find- ing in this disease, and the concentration of serum copper is of greater fluctuation than in control subjects, an attempt has been made â??to eva- luate the activity of ceruloplasmin in multiple sclerosis. The estimation of ceruloplasmin level may be important for better understanding of copper metabolism in multiple sclerosis. The copper plays a part in hio- logical respiration of myelin sheaths. I t is worth noting that in a previous paper on enzymatic activity of some transaminases and copper oxidase among patients with multiple sclerosis, in 3 cases lower levels of ceruloplasmin than in healthy sub- jects have been found, Cendrowski (1961). ....Learn more/transcript: https://dokumen.tips/documents/the-oxidase-activity-of-serum-ceruloplasmin-in-multiple-sclerosis.html
THE OXIDASE ACTIVITY OF SERUM CERULOPLASMIN IN MULTIPLE SCLEROSIS
Click on the article title to read more.
Saturday, September 23, 2017 11:01 PM | CCSVI in Multiple Sclerosis shared Canadian Neurovascular Health Society's post. Volg link

Canadian Neurovascular Health Society
We are now live tweeting from our conference! Follow CNH_Society for the latest!

https://twitter.com/cnh_society/status/911691024652582912
Friday, September 22, 2017 5:52 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Episode 152: Iron Toxicity and Magnesium

In this episode you will learn:
• How bioavailable copper prevents iron toxicity damage in your body
• The hidden danger of iron supplements
• The critical role magnesium plays in your health that nobody is talking about

Morley had been a hospital executive and consultant for 32 years until he developed a condition called “frozen shoulder.” Which opened his eyes to the power of natural medicine, and set him on his path of becoming an expert in the interplay between magnesium, copper and iron metabolism.

Through his Magnesium Advocacy Group (MAG), he is committed to educating as many people as possible about the importance of iron toxicity and how magnesium and copper help regulate the well-being of all.
http://lymeninjaradio.com/152-morley-robbins/
#152: Morley Robbins, The Magnesium Man – Iron Toxicity and Magnesium
Morley had been a hospital executive and consultant for 32 years until he developed a condition called “frozen shoulder.” Which opened his eyes to the power of natural medicine, and set him on his …
Thursday, September 21, 2017 5:24 PM | Nigel Wadham Volg link
"“This all comes down to immune system and brain interaction,” said Kipnis, chairman of UVA’s Department of Neuroscience. “The two were believed to be completely not communicating, but now we’re slowly, slowly filling in this puzzle. Not only are these [immune] cells present in the areas near the brain, they are integral to its function. When the brain is injured, when the spinal cord is injured, without them, the recovery is much, much worse.”

Curiously, the immune cells were found along the vessels discovered by Kipnis’ team. “They’re right on the lymphatics, which is really weird,” noted researcher Sachin Gadani. “You have the lymphatics and they’re stacked right on top. They’re not inside of them – they’re around them.”"
Characterization of meningeal type 2 innate lymphocytes and their response to CNS injury
The meningeal space is occupied by a diverse repertoire of immune cells. Central nervous system (CNS) injury elicits a rapid immune response that affects neuronal survival and recovery, but the role of meningeal inflammation remains poorly understood. Here, we describe type 2 innate lymphocytes (ILC...
Wednesday, September 20, 2017 7:14 PM | CCSVI in Multiple Sclerosis Volg link
2017 VEITHsymposium
Saturday, November 18, 2017
Update On The Endovascular Treatment Of Chronic Cerebrospinal Venous Insufficiency (CCSVI) To Improve Multiple Sclerosis Symptoms: Status And Results Of The BRAVE DREAMS Randomized Sham Controlled Trial
CCSVI in Multiple Sclerosis
Wednesday, September 20, 2017 6:23 PM | CCSVI in Multiple Sclerosis Volg link
The NEURO program for brain health: Nutrition, Exercise, Unwind, Restorative Sleep, and Optimize Mental and Social Activity. For readers of this page, this new program will sound familiar! <3
#NotAPill #HeartBrainConnection
Researchers Say Most Alzheimer's Disease Cases Are Preventable—Find Out How - Blue Zones
More than five million Americans have Alzheimer’s disease and some experts forecast that those numbers will triple by 2050. Drug companies have spent billions of dollars trying to understand disease and develop a cure or effective treatments, but they have been largely unsuccessful. Ayesha Sherzai,…
Wednesday, September 20, 2017 5:54 PM | CCSVI in Multiple Sclerosis shared Canadian Neurovascular Health Society's note. Volg link
Tix still available! Incredible line up of speakers and information on ways to improve neurovascular health.
Canadian Neurovascular Health Society
Tuesday, September 19, 2017 9:51 PM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Dr. Zamboni via Twitter: "...A Canadian article linking #CCSVI-microbleeds-heme iron-freeradicals-myelin injury #MultipleSclerosis..."

'Potential role of ferric hemoglobin in MS pathogenesis: Effects of oxidative stress and extracellular methemoglobin or its degradation products on myelin components.' Free Radic Biol Med. 2017 Aug

Abstract
There is a well-documented relationship between cerebral vasculature and multiple sclerosis (MS) lesions: abnormal accumulations of iron have been found in the walls of the dilated veins in cerebral MS plaques. The source of this iron is unknown, but could be related to the recognized phenomenon of capillary and venous hemorrhages leading to blood extravasation. In turn, hemorrhaging leading to hemolysis results in extracellular release of hemoglobin, a reactive molecule that could induce local oxidative stress, inflammation, and tissue damage. Our previous studies with a reduced form of hemoglobin (oxyHb) have demonstrated its ability to cause extensive lipid and protein oxidation in vitro, which would result in membrane destabilization. Here, we investigated in further detail the mechanism by which the more abundant oxidized form of extracellular hemoglobin (metHb), and dissociated hemin, cause direct oxidative damage to myelin components, specifically membrane-mimetic lipid vesicles and myelin basic protein (MBP), a highly-abundant protein in the CNS. Oxidation of lipids was assessed by the formation of conjugated diene/triene and malondialdehyde, and oxidation of MBP was demonstrated by the bityrosine formation and by the change in protein mass. Our results show that metHb causes oxidative damage to MBP and myelin lipids, partly by transferring its hemin moiety to protein and lipid, but mostly as an intact protein possibly via formation of a ferryl radical. These results elucidating the mechanism of extracellular hemoglobin-induced oxidative damage to myelin components support the need for further research into vascular pathology in MS pathogenesis, to gain insight into the role of iron deposits and/or in stimulation of different comorbidities associated with the disease.

https://www.ncbi.nlm.nih.gov/pubmed/28863941
Venöse Multiple Sklerose, CVI & SVI, CCSVI
Tuesday, September 19, 2017 6:40 PM | CCSVI in Multiple Sclerosis Volg link
Dear MS Societies--Now that the truth is coming out with "Living Proof", you are suddenly recommending lifestyle changes to help MS. Maybe you might want to apologize to Dr. Swank and MS patients for the decades of discrediting his research? Here, I wrote the letter for you.
An Apology Letter to Dr. Roy Swank from the National MS Society
Dear Dr. Roy Swank, We would like to apologize. Remember how we denigrated your cardiovascular health program for people with MS because ...
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