Sunday, May 8, 2016 6:28 PM
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Venöse Multiple Sklerose, CVI & SVI, CCSVI
Iron Chelates and Unwanted Biological Oxidations, by Kevin D. Welch and Steven D. Aust, Department of Chemistry and Biochemistry Biotechnology Center, Utah State University "...Conclusions • The reactivity of iron is highly dependent upon its ligand environment. Any change in the ligand environment of iron, such as pH, buffer, or chelator, will effect its reactivity. • Biomolecules do not autoxidize. Their oxidation is mediated by transition metals such as iron. • Iron can mediate the oxidation of DNA and protein. DNA and most likely protein are oxidized by the hydroxyl radical. • Iron appears to have a direct effect on the oxidation of lipids. Lipids can be oxidized by the hydroxyl radical but some type of Fe(II)-O2- Fe(III) complex also promotes lipid peroxidation. The iron-mediated oxidations of DNA, protein, lipid, and organic chemicals do not occur via the same mechanism." learn more: http://www.sfrbm.org/frs/AustIronChelates.pdf
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