One of our CCSVI friends from Italy did send me a very interesting link/information. Thanks Francesco Paolo Ruggieri! http://www.youtube.com/watch?v=KoeaU-iinww&feature=player_embedded
“Following is the point of view of a doctor, vascular surgeon, who has been exposed to my ‘case’. I value he has formulated a ‘dignified’ hypothesis and a very measured one too which deserves being looked into. Finally someone replies to me!!

«Good morning Antonello, I had time to think and will briefly summarize what my deductions are, simply based on logic and exclusion criteria; obviously every theory must be demonstrated well in other ways.
Moreover, my thoughts want to be purposely associated to the CCSVI theory of which we have extensive knowledge, they should only be associated with events relative our modest experience.
To begin with, the vascular return is, generally, alimented by four principal factors that highly vary depending on body areas that are considered. These factors are
1) the force driving the venous return of peripheral blood given by the cardiac pump
2) the thorax negative pressure given by the respiratory actions
3) the presence of the muscular pump
4) the force of gravity

In the particular case of cerebro-spinal and inter-cranium vein circulation, in general, the forces are reduced to 3. This excludes the muscular pump at this level. The force of gravity performs its regular action as our trials are performed in an erect posture.

At this point, there are only 2 variables left to which we must pay attention:
1) the cardiac pump
2) the negative inter-thorax pressure

These 2 variables are the ones that change in the physical exercise, and in our case, with an increase of cardiac frequency during breathing.
The increase in frequency associated to concomitant positive inotropism makes it possible for increase in the force driving the venous return of peripheral blood given by the cardiac pump from the artery torrent to the vein torrent.
The increase of the frequency during breathing and their ‘depth’ influence the significant increase of vein return in the right ventricle.
In simple terms, this translates in an increase in vein reflux from the cerebrum-spinal torrent towards the heart, both if this happens through regular calibre veins and if this happens with restricted calibre veins, similarly as it happens with hyraulic pumps with tight diameter – by increasing the positive pressure at the highest point or increasing the negative pressure at the lowest point.
At the cerebral-spinal parenchyma level, this causes a momentary reduction of the perilesional or interlesional endema in a momentary way, conversely as a PTA causes it in a lasting way and a stent (the correct one) would cause it in a steady way.

From this derives, in our case, a momentary resolution of the symptoms, in the PTA case, a more lasting resolution and in the case of the stent (the correct one) a steady resolution of the symptom.
The cold temperature is a secondary case which could act by increasing the vein return by peripheral vascular restriction, but has a percentage of importance which must be evaluated..
These are only theories based on impressions; but they could be the start to confirm or not.. lets not forget that we could be playing with a bioumoral theory which is completely disconnected form what we considered above and what I have written could only be, as it often happens, given by the fact that sometimes one falls in love with one’s son and one doesn’t want to look further…
To someone else the responsibility to continue on the topic…

With warmth, Franco» “http://nuotoesclerosimultipla.blogspot.it/
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CCSVI e Sclerosi Multipla
"liberazione" temporanea? Contatti: antonello.tovo@gmail.com http://nuotoesclerosimultipla.blogspot.com/