Please read this note I posted last January about hypoperfusion (slow blood flow) and the MS brain.  Many researchers had noted this phenomena before Dr. Zamboni came forward with his discovery of CCSVI.  Slow blood can create an hypoxic (low oxygen) situation in the brain, and this in itself can damage brain tissue, causing axonal death and activating the immune system.  Here is the note:

http://www.facebook.com/note.php?note_id=229656952210

Remember, the autoimmune theory of MS is still just a theory.  Doctors have never proven that MS is started by the immune system.  

Here is a wonderful editorial on this topic by Dr. Peter  Behan, called

"The Futility of the autoimmune orthodoxy in multiple sclerosis research"

"...a false orthodoxy claiming that multiple sclerosis is an autoimmune disorder has developed and formed the present basis of treatment, drug trials and research. The outcome of this misplaced creed has been truly catastrophic.”

http://www.expert-reviews.com/doi/pdf/10.1586/ern.10.69

If you are curious as to how CCSVI could cause lesions and brain and spinal damage in MS, please, read this note and the paper I have linked.  Yes, it is very technical, but I break it down into chunks, and explain what the researchers found.

Again, Here's the Note

http://www.facebook.com/note.php?note_id=229656952210

No matter what neurologists may claim, they have never proven that MS is a purely autoimmune driven disease.  Researchers have noted that in the beginning the lesions look like ischemic (low oxygen) events, even before the immune system is activated.   Here is a link to Lassmann's paper on this:  http://www.ncbi.nlm.nih.gov/pubmed/12559509

Here is a paper by Prineas and Barnett--where they study fresh lesions upon autopsy, and discover that there is axonal death without ANY immune activation:  This discovery makes them question EAE as a model for MS.

Relapsing and Remitting Multiple Sclerosis: Pathology of the Newly Forming Lesion 

Michael H. Barnett, MBBS and John W. Prineas, MBBS 

The study describes the clinical and pathological findings in 12 patients with relapsing and remitting multiple sclerosis, 

who died during or shortly after the onset of a relapse. Pathological changes not previously associated with the formation of new symptomatic lesions were observed in seven cases, namely, extensive oligodendrocyte apoptosis and microglial activation in myelinated tissue containing few or no lymphocytes or myelin phagocytes. No current laboratory model of multiple sclerosis, in particular, experimental allergic encephalomyelitis, is known with these features, which raises the possibility of some novel process underlying new lesion formation in multiple sclerosis. 

http://www.cpnhelp.org/files/Ref1_Annals04.pdf

It is vitally important that we understand the science behind Dr. Zamboni's discovery, in order to be informed patients and caregivers.  Slowed venous drainage can create slowed perfusion....just like all those researchers noted in MS brains.  There will continue to be much push  back from the status quo.   They need to maintain the current dogma on MS, in order to keep their jobs and pharmaceutical ties.   But we need to ask, How do you KNOW MS is initiated by the immune system?  Have you read the other research??  

Be informed.  Read the research.  It's not snake oil.  It's scientific fact.

http://www.facebook.com/note.php?note_id=439394977210&id=110796282297