Magnesium and Concussive Brain Injury
[The Neurometabolic Cascade of Concussion]

SUMMARY
Cerebral concussion is followed by a complex cascade of ionic, metabolic, and physiologic events. The earliest changes are an indiscriminate release of EAAs and a massive efflux of K+, triggering a brief period of hyperglycolysis. This is followed by more persistent Ca2+ influx, mitochondrial dysfunction with decreased oxidative metabolism, diminished cerebral glucose metabolism, reduced CBF, and axonal injury...“ Journal of Athletic Training, 2001

Full paper: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC155411/