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Saturday, October 21, 2017 10:35 PM | Venöse Multiple Sklerose, CVI & SVI, CCSVI shared Canadian Neurovascular Health Society's post. Volg link
How the new impatient patient is disrupting medicine
Patient activists have never been as vocal. But are they truly being heard?
Anne Kingston, October 21, 2017


"...Chatrooms and Facebook pages became places to mobilize, to circulate petitions, and to exchange research that ran contrary to medical dogma.
That was the story of CCSVI, which saw a non-doctor communicate directly with a doctor willing to perform a procedure outside of the MS mainstream. Joan Beal, a Los Angeles-based opera singer, began investigating MS online in 2007 after her husband, music composer Jeff Beal, was diagnosed...."
Canadian Neurovascular Health Society
http://www.macleans.ca/society/health/how-the-new-impatient-patient-is-disrupting-medicine/
Saturday, October 21, 2017 6:29 PM | CCSVI Alliance Volg link
"Understanding a role for hypoxia in lesion formation and location in the deep and periventricular white matter in small vessel disease and multiple sclerosis"
https://www.ncbi.nlm.nih.gov/pubmed/29026001

(An excellent resource to learn about hypoxia in MS is Dr. Philip James's book "Oxygen and the Brain". http://www.hbot.com/blog/edward-lucarini/oxygen-and-brain-dr-philip-james-explains-relationship-his-new-book)
Understanding a role for hypoxia in lesion formation and location in the deep and periventricular white matter in small vessel disease and multiple... - PubMed - NCBI
Clin Sci (Lond). 2017 Oct 12;131(20):2503-2524. doi: 10.1042/CS20170981. Print 2017 Oct 15. Review
Friday, October 20, 2017 1:23 AM | CCSVI in Multiple Sclerosis Volg link
Interview on Brain Drain on BBC. Begins at 1:00 minute in. Daniel Reich talks about this discovery in humans. As the medical reporter later says, "making sure a drainage system is flowing properly can be really important for the health of the brain..." Yes, that's pretty much what we've been saying on this page for eight years.
Discovery of Brain’s “Waste Disposal” System, Health Check - BBC World Service
Scanning reveals brain’s “waste disposal” system
Thursday, October 19, 2017 6:02 PM | CCSVI Alliance Volg link
Jonathan Kipnis tweets:

(Podcast from the BBC - interview with Daniel Reich from the NIH on the brain's waste removal system. Interview starts at 01:00 - it is about 7 minutes in length)
http://www.bbc.co.uk/programmes/w3csty7t
CCSVI Alliance
Thursday, October 19, 2017 12:04 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
'Anemia of Chronic Inflammation' is the OPPOSITE of 'Iron Deficiency Anemia'. Get PROPERLY tested before killing yourself with highly toxic isolated Iron supplements! http://requestatest.com/mag-zinc-copper-panel-with-iron-panel-testing
Venöse Multiple Sklerose, CVI & SVI, CCSVI
Tuesday, October 17, 2017 11:49 PM | CCSVI in Multiple Sclerosis Volg link
Recent research on MS, white matter lesions and hypoxia (or a lack of oxygen in the brain) is not new. There are studies going back decades that made this connection to the vasculature. Here's more, in a post from 2009.
Vascular white matter lesions are 50-500 times more likely than MS
December 30, 2009 at 2:57pm Neurologists refer to the "unique" presentation of MS as an autoimmune disease- with oligoclonal banding...
Tuesday, October 17, 2017 4:44 PM | CCSVI in Multiple Sclerosis Volg link
Dr. Zamboni shares new research from London--hypoperfusion and hypoxia are related to lesions in #MS. Vascular connection is real. #HeartBrainConnection https://www.ncbi.nlm.nih.gov/pubmed/29026001
Understanding a role for hypoxia in lesion formation and location in the deep and periventricular white matter in small vessel disease and multiple... - PubMed - NCBI
Clin Sci (Lond). 2017 Oct 12;131(20):2503-2524. doi: 10.1042/CS20170981. Print 2017 Oct 15. Review
Tuesday, October 17, 2017 7:17 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Understanding a role for hypoxia in lesion formation and location in the deep and periventricular white matter in small vessel disease and multiple sclerosis. Clin Sci (Lond). 2017 Oct

Abstract
The deep and periventricular white matter is preferentially affected in several neurological disorders, including cerebral small vessel disease (SVD) and multiple sclerosis (MS), suggesting that common pathogenic mechanisms may be involved in this injury. Here we consider the potential pathogenic role of tissue hypoxia in lesion development, arising partly from the vascular anatomy of the affected white matter. Specifically, these regions are supplied by a sparse vasculature fed by long, narrow end arteries/arterioles that are vulnerable to oxygen desaturation if perfusion is reduced (as in SVD, MS and diabetes) or if the surrounding tissue is hypoxic (as in MS, at least). The oxygen crisis is exacerbated by a local preponderance of veins, as these can become highly desaturated 'sinks' for oxygen that deplete it from surrounding tissues. Additional haemodynamic deficiencies, including sluggish flow and impaired vasomotor reactivity and vessel compliance, further exacerbate oxygen insufficiency. The cells most vulnerable to hypoxic damage, including oligodendrocytes, die first, resulting in demyelination. Indeed, in preclinical models, demyelination is prevented if adequate oxygenation is maintained by raising inspired oxygen concentrations. In agreement with this interpretation, there is a predilection of lesions for the anterior and occipital horns of the lateral ventricles, namely regions located at arterial watersheds, or border zones, known to be especially susceptible to hypoperfusion and hypoxia. Finally, mitochondrial dysfunction due to genetic causes, as occurs in leucodystrophies or due to free radical damage, as occurs in MS, will compound any energy insufficiency resulting from hypoxia. Viewing lesion formation from the standpoint of tissue oxygenation not only reveals that lesion distribution is partly predictable, but may also inform new therapeutic strategies.

https://www.ncbi.nlm.nih.gov/pubmed/29026001
Venöse Multiple Sklerose, CVI & SVI, CCSVI
Tuesday, October 17, 2017 4:18 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Nothing Boring About Boron

"...Conclusions

Boron has been proven to be an important trace mineral because it (1) is essential for the growth and maintenance of bone; (2) greatly improves wound healing; (3) beneficially impacts the body’s use of estrogen, testosterone, and vitamin D; (4) boosts magnesium absorption; (5) reduces levels of inflammatory biomarkers, such as hs-CRP and TNF-a; (6) raises levels of antioxidant enzymes, such as SOD, catalase, and glutathione peroxidase; (7) protects against pesticide-induced oxidative stress and heavy-metal toxicity; (8) improves brain electrical activity, cognitive performance, and short-term memory in elders; (9) influences the formation and activity of key biomolecules, such as SAM-e and NAD+; (10) has demonstrated preventive and therapeutic effects in a number of cancers, such as prostate, cervical, and lung cancers and multiple and non-Hodgkin’s lymphoma; and (11) may help ameliorate the adverse effects of traditional chemotherapeutic agents. Americans’ daily dietary intake of boron was estimated to be approximately 1 mg/d in 1999.

In none of the numerous studies conducted to date, however, do boron’s beneficial effects appear at intakes of less than 3 mg/d. No EARs or DRIs have been set for boron; only a UL of 20 mg/d for individuals aged 18 years or older. The absence of studies showing harm in conjunction with the substantial number of articles showing benefits support the consideration of boron supplementation of 3 mg/d for any individual who is consuming a diet low in fruits and vegetables or who is at risk for or has osteopenia; osteoporosis; OA; or breast, prostate, or lung cancer...."
full paper: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4712861/#!po=66.9643
Venöse Multiple Sklerose, CVI & SVI, CCSVI
Sunday, October 15, 2017 9:21 PM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Magnesium protects cognitive functions and synaptic plasticity in streptozotocin-induced sporadic Alzheimer's model., 2014

"...We conclude that magnesium treatment protects cognitive function and synaptic plasticity by inhibiting GSK-3ß in sporadic AD model rats, which suggests a potential role for magnesium in AD therapy...

...In summary, we found that simultaneous intraperitoneal injections of magnesium sulfate significantly enhanced the brain magnesium levels, improved synaptic efficacy, and prevented memory and learning impairments through modifications of synaptic proteins and Tau phosphorylation in ICV-STZ rats. Our findings provide novel insights suggesting that magnesium treatment at the early stage may decrease the risk for cognitive impairment in AD."
Full paper: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4182554/
Venöse Multiple Sklerose, CVI & SVI, CCSVI
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