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Wednesday, October 17, 2018 7:32 PM | Venöse Multiple Sklerose, CVI & SVI, CCSVI shared Canadian Neurovascular Health Society's post. Volg link
Traditional Uses of Cannabinoids and New Perspectives in the Treatment of Multiple Sclerosis, 15 August 2018

Abstract: Recent findings highlight the emerging role of the endocannabinoid system in the control of symptoms and disease progression in multiple sclerosis (MS). MS is a chronic, immune-mediated, demyelinating disorder of the central nervous system with no cure so far. It is widely reported in the literature that cannabinoids might be used to control MS symptoms and that they also might exert neuroprotective effects and slow down disease progression. This review aims to give an overview of the principal cannabinoids (synthetic and endogenous) used for the symptomatic amelioration of MS and their beneficial outcomes, providing new potentially possible perspectives for the treatment of this disease.
full paper: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6164967/pdf/medicines-05-00091.pdf
Canadian Neurovascular Health Society
Hoping this new era brings broader awareness and less stigmatism while ensuring greater availability to those who need cannabis for medical purposes. ????

A reminder of its benefits:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6164967/
Wednesday, October 17, 2018 5:43 PM | The Vascular Connection to Multiple Sclerosis Volg link
Molecular MRI of CNS endothelial cells is now a reality. This new technology allows researchers to see the process behind the break-down of the blood brain barrier. "Upon injury, the endothelial cells shift from a quiescent to an activated phenotype, involving increased vascular permeability, switch to a pro-thrombotic state, production of pro-inflammatory cytokines and exposure of adhesion molecules." #VascularConnectiontoMS #EndothelialHealthProgram
Molecular Magnetic Resonance Imaging of Endothelial Activation in the Central Nervous System
Endothelial cells of the central nervous system over-express surface proteins during neurological disorders, either as a cause, or a consequence, of the disease. Since the cerebral vasculature is easily accessible by large contrast-carrying particles, ...
Wednesday, October 17, 2018 5:24 PM | The Vascular Connection to Multiple Sclerosis shared Gladstone Institutes's post. Volg link
“Our study supports that vascular damage leading to immune-driven neurodegeneration may be a common thread between diseases of different etiologies with blood-brain barrier leaks,” says Akassoglou."
Microbleeds harm the brain-- causing iron deposition, oxidative stress, inflammation, and neuronal death. Let's learn why these microbleeds happen in #MS, and find the root cause.
Gladstone Institutes
Researchers led by Gladstone neuroscientist Katerina Akassoglou are seeking better treatments for neurological disorders linked to inflammation in the brain, such as Alzheimer’s disease and multiple sclerosis.
Tuesday, October 16, 2018 11:55 PM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
ALL, I repeat, ALL ATP Pumps are regulated by Mg-ATP! Without Magnesium there is no energy, no life.

“Importance of magnesium for the electrolyte homeostasis - an overview, by Armin Schroll, Deutsches Herzzentrum München, Germany

Summary: Disorders of electrolyte homeostasis are known at many diseases and clinical situations. They have serious consequences for the cell. Mg-deficiency is followed by a K-deficiency, which cannot be equalized by K alone: a refractory hypokalemia always needs additional Mg supply for its restitution. From K, Mg-deficiency a Na/Ca-overload of the cell with aggravating consequences will follow: impaired activity and vitality with electric instability. Mg, which is responsible for development of a Ca-overload is also able to restore electrolyte homeostasis by sufficient supply competitively. The pathophysiologic relations for development of a cellular imbalance and its restitution concern the Na/K-pump, the Ca-pump and the Na/Ca-exchange. The clinical applications of Mg therefore are manifold: recovery under diuretic treatment, coronary heart disease, arrhythmias, perioperative electrolyte therapy, transcellular shifts, coronary dilatation and so on. ..”

Learn more/full paper: http://www.mgwater.com/schroll.shtml
Magnesium and Electrolyte Homeostasis
Disorders of electrolyte homeostasis are known at many diseases and clinical situations. They have serious consequences for the cell. Mg-deficiency is followed by a K-deficiency, which cannot be equalized by K alone: a refractory hypokalemia always needs additional Mg supply for its restitution. Fro...
Tuesday, October 16, 2018 1:25 AM | The Vascular Connection to Multiple Sclerosis shared Gladstone Institutes's post. Volg link
Why is blood getting into the #MS brain in the first place? #fibrin #coagulationcascade #bloodbrainbarrier http://ccsviinms.blogspot.com/2014/03/blood-matters.html
Gladstone Institutes
#AkassoglouLab developed an antibody to stop the detrimental effects of blood-brain barrier leaks and protect multiple sclerosis and Alzheimer’s disease models.
Sunday, October 14, 2018 11:10 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Cannabis is Healing the Injured Brain – Here’s the Latest
https://www.rxleaf.com/post/7132/cannabis-is-healing-the-injured-brain-heres-the-latest
Venöse Multiple Sklerose, CVI & SVI, CCSVI
Monday, October 8, 2018 11:44 PM | CCSVI Alliance Volg link
"Physiotherapy treatment for muscular compression"
- Physiotherapy and osteopatical approach, releasing vertebrae and bone fragments where these
muscles are attached, giving it back their proper functionality, just as much as to relevant
metameres innervation
- Fascial approach starting from the myofascial tissue shall be carried out manoeuvring of pompage,
overstretching and mild compression of the muscle tissues until a feeling of their re-lease
- Both approaches should be implemented with repositioning methods on the planes
(sag-ittal, coronal, transverse).

https://www.pagepressjournals.org/index.php/vl/article/view/7760/7453
The omohyoid and sternocleidomastoid muscles entrapment of the internal jugular vein: Which role in Mèniére disease patients? Treatment perspective description | Piraino | Veins and Lymphatics
The omohyoid and sternocleidomastoid muscles entrapment of the internal jugular vein: Which role in Mèniére disease patients? Treatment perspective description
Friday, October 5, 2018 7:07 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Magnesium and Concussive Brain Injury
The Neurometabolic Cascade of Concussion.

http://www.nutritionalmagnesium.org/magnesium-and-concussive-brain-injury/
Magnesium and Concussive Brain Injury | Nutritional Magnesium Association
The average American diet, government studies show us, provides only 40%—less than half the magnesium that we all need in a day. So almost everybody is low!Sherry A. Rogers, M.DAuthor of Wellness Against All Odds
Friday, October 5, 2018 6:51 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Use of magnesium in traumatic brain injury.

Abstract

Depletion of magnesium is observed in animal brain and in human blood after brain injury. Treatment with magnesium attenuates the pathological and behavioral changes in rats with brain injury; however, the therapeutic effect of magnesium has not been consistently observed in humans with traumatic brain injury (TBI). Secondary brain insults are observed in patients with brain injury, which adversely affect clinical outcome. Systemic administration studies in rats have shown that magnesium enters the brain; however, inducing hypermagnesemia in humans did not concomitantly increase magnesium levels in the CSF. We hypothesize that the neuroprotective effects of magnesium in TBI patients could be observed by increasing its brain bioavailability with mannitol. Here, we review the role of magnesium in brain injury, preclinical studies in brain injury, clinical safety and efficacy studies in TBI patients, brain bioavailability studies in rat, and pharmacokinetic studies in humans with brain injury. Neurodegeneration after brain injury involves multiple biochemical pathways. Treatment with a single agent has often resulted in poor efficacy at a safe dose or toxicity at a therapeutic dose. A successful neuroprotective therapy needs to be aimed at homeostatic control of these pathways with multiple agents. Other pharmacological agents, such as dexanabinol and progesterone, and physiological interventions, with hypothermia and hyperoxia, have been studied for the treatment of brain injury. Treatment with magnesium and hypothermia has shown favorable outcome in rats with cerebral ischemia. We conclude that coadministration of magnesium and mannitol with pharmacological and physiological agents could be an effective neuroprotective regimen for the treatment of TBI. https://www.ncbi.nlm.nih.gov/pubmed/20129501
Venöse Multiple Sklerose, CVI & SVI, CCSVI
Thursday, October 4, 2018 12:25 AM | Venöse Multiple Sklerose, CVI & SVI, CCSVI Volg link
Venous insufficiency: Differences in the content of trace elements. A preliminary report, Adv Clin Exp Med.
2018

“...Discussion
Similar research has already been done, but there is very little literature available for comparison. The pioneer study of ferrous content in veins was performed by Krzysciak et al. (6) One of the mechanisms involved in chronic venous disease is the destructive effect of free oxygen and free nitrogen radicals. The authors showed that Fe ions were involved in the oxidative damage mechanism, which caused tissue altering via the Fenton reaction. Oxidative stress correlated with insufficiency in the venous tissue, and increased SOD activity was observed compared to normal venous tissue. (15) An increased level of iron in the skin of patients with chronic venous disease was described by Myers in 1966. (43) Degradation of hemoglobin, as well as Fe release, was associated with erythrocyte dysfunction. Moosavi et al. showed increased concentrations of iron and copper in the walls of varicose veins compared to controls, using a proton-induced X-ray emission (PIXE) analysis...”

“...Conclusions
This paper shows the levels of certain ions in insufficient and normal veins, particularly the differences between the concentrations of Mg, Mn, Ca, and Si. This study is preliminary in nature, but the data obtained suggests that the oxidative damage mechanism is involved in the development of varicose veins and chronic venous insufficiency...”

Full paper: http://www.advances.umed.wroc.pl/pdf/2018/27/5/695.pdf
Venöse Multiple Sklerose, CVI & SVI, CCSVI
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